Why fluoxetine?
In addition, after cured myelotoxic agranulocytosis, the body becomes more resistant to this type of drug, and a large dose will be needed to cause disturbances, and in the case of fluoxetine agranulocytosis, on the contrary, pathological reactions will develop even after extremely low test doses.Another specific difference of hapten agranulocytosis is its acute nature - with the timely cancellation of the drug, which has become a hapten, the number of granulocytes in the blood is restored relatively quickly. Haptenic agranulocytosis can develop at any age, but it rarely occurs in children. It is believed that women develop this complication of prozac drug therapy more often than men, but with age, this difference is leveled.

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Cohort 2
Class of 2019
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prozac the fact that chlorpromazine is considered a drug capable of causing myelotoxic agranulocytosis in persons with idiosyncrasy (with special sensitivity to the drug), in many patients, using special tests for antibodies, the immune nature of agranulocytosis was revealed.
12.16.2021
In other patients, an autoimmune reaction developed with the formation of antibodies to the protein elements of the nuclei of leukocytes, as is the case, for example, with systemic lupus erythematosus. Of course, in such cases, agranulocytosis developed gradually, and had a tendency to a chronic course. Thus, the mechanism of development of drug agranulocytosis depends not only on the nature of the drug, but also on many other factors, such as the gender and age of the patient, the state of his immune system, the presence and severity of concomitant diseases, etc.
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In such patients, the clinical picture of drug agranulocytosis developed rapidly and violently, but discontinuation of the drug gave a fairly quick positive result.
01.12.2021
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